Tag Archives: animals
SIGNS OF THE APOCALYPSE – 500 dead penguins wash up along Brazil’s beaches
- Hundreds wash ashore in Rio Grande do Sul, biologists say
- Birds are decomposed but otherwise seem unharmed
- Penguins migrate from Argentina about this time of year, biologist says
(CNN) — Marine biologists in Brazil have launched an investigation after hundreds of dead penguins washed ashore in the southern state of Rio Grande do Sul, they said Monday.
More than 500 birds have been found on beaches over the past week, and that figure has steadily risen over the past few days, according to experts from Brazil’s Center of Coastal and Marine Studies (Ceclimar).
Japan’s fugitive penguin captured
The figure has been especially puzzling for the marine scientists, who say the birds are badly decomposed but otherwise seem unhurt and without oil stains.
The discovery follows an official inquiry launched by the Peruvian government this year when close to 3,000 dolphins and more than 500 pelicans washed up along the country’s northern coast.
Mauricio Tazeres, a biologist from the Center of Coastal and Marine Studies, said “it’s actually pretty common for us to find at least some penguins but never in this number.
Stolen penguin back safe at Sea World
“The animals usually migrate from Argentina around this time of year in search of food and warmer weather, and each year, some do wash up. But over 500 is a very, very high number, and right now, I simply do not have an explanation.
“We have collected some samples for autopsy, but the animals are so decomposed, it is going to make analysis very difficult,” he said. “The animals were a lot smaller in terms of size and weight than normal penguins, so we think it will be natural causes, but it is certainly very strange.”
IF YOU THOUGHT ZOMBIES WHERE TOUGH – Try Surviving Yellowstone’s super volcano
For preppers, it’s the ultimate end game: surviving the eruption of Yellowstone’s super volcano.
While some folk worry about an asteroid strike bringing about the end of the world as we know it, as scientists say it did for the dinosaurs 65 million years ago, North America is actually sitting on its own extinction-level event waiting to happen.
“Everything would be wiped out; it would take years for the climate to recover and decades for the rivers to clear up because everything would be choked with volcanic ash for a wide area around the eruption site,” said Kelly Russell, professor of volcanology at the University of British Columbia. “The southern latitudes of Alberta, Saskatchewan and Manitoba would all see ash cover, how thick it would be depends on the winds and the amount of magma.”
Russell stresses that such super eruptions are extremely rare — the last one happened before human civilization — but that they can and do happen, and Yellowstone, in Yellowstone National Park in Wyoming, is an active field that has seen three massive eruptions.
He compares Yellowstone to Mount St. Helens in Washington state in 1980.
“It produced one cubic kilometre of magma, and we saw ash fall from it in southern B.C., a small amount, but it was there, and when we look at the very largest eruptions that have taken place at Yellowstone, they can spew out a thousand cubic kilometres, so that’s a thousand times larger than the Mount St. Helens eruption that’s in everybody’s minds.”
Russell points out that two feet of ash from the Crater Lake eruption in Oregon 7,700 years ago can be found in Oliver, B.C., and in the banks of the Bow River in Calgary. He says if Yellowstone cuts loose, the southern Canadian prairies could get covered in many feet of ash, and the American states closest to Yellowstone would be smothered with an even thicker layer of the sterile rock powder, killing off livestock and leaving them unable to grow food.
“The ash is terrible, take a window and grind it into a coarse flour, then breath that in, it does terrible things to the human body. It would be important to have masks and filters, if you were trying to survive it,” Russell said. “The United States would stop being a food-exporting nation and starting being a food-importing nation.”
Yellowstone’s volcano doesn’t have the classic menacing cone shape, so many people don’t know that a magma chamber bigger than New York City lies beneath the steaming surface.
640,000 years ago, animals similar to elephants, rhinoceroses and zebras roamed the plains of the United States when Yellowstone blew — and it took out the animals and every other living thing that couldn’t fly away from the blast.
Scientists estimate Yellowstone’s volcano explodes every 600,000 to 700,000, and some say the time could be coming for another eruption. If it blows, the chances of survival sound bleak, but that’s not stopping some survivalists from preparing.
“Some people who visit there say there are more hot springs popping up there, and there is more of a sulphur smell that’s stronger than ever. You take from it what you want and I just pay attention to it a little more than the average person, just in case,” said Jason Charles, a firefighter in New York City. “I don’t want it to be a curve ball we don’t see coming. I keep it in the back of my head.”
Charles was a paramedic on 9/11 and saw thousands of people struggling to breath through the toxic dust. He has special dust and ash filters for his gas mask in case of another NYC disaster, or if Yellowstone blows.
“I know some people who have bought UV lights, in case they need to grow their own food – but that’s also assuming that the power grid stays up, but then there’s a space issue, how much could you grow?” he said. “It’s better to store food that will last.”
Charles has a one-year supply of food for his family, including his wife and four children. Meal-ready-to-eats (MREs), canned pasta and lots of canned fruit are stuffed into his apartment and his storage locker. He prepares for all sorts of disasters because he says no one should depend on the government to save them.
“The government couldn’t handle (Hurricane) Katrina. Compared to Yellowstone, Katrina was a drop in the bucket,” he said. “I have always heard as a rumour, they would wait for the masses to die and whatever survivors are left, that’s who they’d take care of, because they can’t take care of hundreds of millions of people we have living here in the United States.”
Jake Lowenstern, a geologist with the U.S. Geological Survey, is the guy in charge of the Yellowstone volcano observatory.
“Worst-case scenario, a super eruption is a thousand cubic kilometres of material gets sent out of the magma chamber. When that happens it’s going to send out a lot of ash, and it circles the globe and changes the climate for years, drops the temperature for a few years,” Lowenstern said.
He said while a super eruption is highly unlikely, and even if it does happen not many would survive, it doesn’t hurt to prep.
“Things you can do to prepare for one kind of disaster are useful for any kind of disaster: lots of food, water, medical supplies and batteries on hand, and for an eruption, add good masks, air filters and weather stripping to keep the ash out of homes.”
Massive eruptions of the past:
— Archeologists say human beings barely survived the last super eruption on Earth. 74,000 years ago, Toba blew up on the Island of Sumatra. It cloaked the planet in sulphur, reducing the temperature by 20 degrees, wiping out growing seasons with snow cover nearly all year and causing mass starvation. Geneticists say the disaster reduced the human population to as few as 1000 people, thus causing the genetic similarity between individuals which is traced to the same time period.
— A volcanic eruption has been blamed for plunging civilization into the Dark Ages and triggering the bubonic plague. In his book Catastrophe, archeological journalist David Keys pinpoints a Krakatoa explosion in 535 AD. He says with the power of two billion Hiroshima bombs it darkened the sky and caused drought and flooding all around the world, including in Northeast Africa, which led to a bumper crop of rodents that passed their infected fleas onto rats and mice on European ships docked for trade off of southern Egypt, beginning the spread of the plague of Justinian.
— In 1815, the Tambora volcano erupted in the East Indies. Global temperatures dropped. Europeans and North Americans called it a “year without summer.” Snow fell in New York in June. Frost was recorded in each of the summer months and crops failed. 200,000 people died in Eastern and southern Europe from starvation and typhus.
— In 1783, Iceland’s Laki Volcano erupted. 9,000 people died in Iceland and thousands more died around the world from crop failure, disease and starvation. Temperatures dropped 13 degrees Celsius.
— Mount Pinatubo erupted in 1991 in the Philippines, a comparably small explosion, ejecting 20 million tonnes of sulphur. Scientists say it reduced global temperatures by about 2 degrees.
CDC visitors exposed to deadly germs?
Washington (CNN) — It’s a highly secured, sophisticated research lab studying deadly diseases such as bird flu, monkeypox, tuberculosis and rabies.
It’s in a facility called Building 18, which cost taxpayers $214 million.
And now, the Biosafety Level 3 lab at the Centers for Disease Control and Prevention in Atlanta is also the subject of a congressional investigation after a potentially dangerous airflow leak at that lab, CNN has learned.
The leak occurred on February 16, when air flowed the wrong way out of a germ lab into a clean-air corridor, rather than through the powerful HEPA filter that cleans the air, congressional sources and CDC officials said. Visitors touring the facility were in the clean corridor when they observed a puff of air being pushed out from the lab through a slot in a door window.
CNN recommends: Deadly bird flu could become airborne
If experiments had been under way at the time of that air leak, experts say, unprotected visitors could have been exposed to deadly germs, although an epidemic would have been unlikely.
According to U.S. Rep. Michael Burgess, a Texas Republican and a medical doctor, the House Energy and Commerce Committee has asked the CDC for documents about that incident. The request came in the wake of a report on internal CDC e-mails about the incident, first reported by USA Today last week.
“The biggest concern was that there was a contingent of visitors who were walking through the building,” Burgess said. “And had one of those people been stricken or made ill or worse, obviously that would have been devastating.”
The lab handles small mammals such as rats, ferrets and mice as part of its experiments with pathogens, according to CDC officials. They say animals were in the lab at the time of the air leak, but they were secured in filtered cages.
CDC officials say the lab was clean, was not active at the time, and no one got infected.
“At no time during recent incidents featured in the media were CDC workers or the public in harm’s way,” agency spokesman Tom Skinner said. “This unique facility features multiple security layers specifically designed to protect workers and the public in the event of an incident.”
In a statement released to CNN, Burgess’ committee said, “We will actively work to find out if there are additional concerns or incidents associated with Building 18. Any anomaly or breach is of concern, and we will work to ensure the integrity of the facility is maintained and that our scientists are safe.”
There has been at least one other safety-related incident in that same building where February’s air leak occurred.
In 2008, it was discovered that a high-containment lab door was sealed with duct tape. That incident was first reported by the Atlanta Journal-Constitution and confirmed to CNN by Skinner.
Robert Hawley, former safety chief at the U.S. Army Medical Research Institute of Infectious Diseases, said the CDC has many safety layers in place at its labs. Hawley says researchers at the Biosafety Level 3 lab work in biosafety “cabinets” within the lab itself.
“Nothing is handled outside that cabinet,” Hawley said. “So they’re working with minute amounts of material, and the chances of aerosol are negligible.”
But there are questions about a possible cover-up.
In an internal e-mail, reported by USA Today, CDC biologist Kismet Scarborough said the centers “… will do anything … to hide the fact that we have serious problems with the airflow and containment in this whole building.”
CNN has not been able to independently verify that e-mail. But in response, Skinner said, “CDC will continue to take an open, transparent and inclusive approach to address any safety challenge in a manner that will ensure the safety of our workforce and the public.”
Skinner said the agency “intends to cooperate fully with Rep. Burgess and the committee to address any questions they may have about Building 18 at CDC.”
Mutant bird flu would be airborne
Mutant bird flu would be airborne, scientists say
Here’s what it takes to make a deadly virus transmissible through the air: as few as five genetic mutations, according to a new study.
This research, published in the journal Science, is the second of two controversial studies to finally be released that examines how the H5N1 bird flu virus can be genetically altered and transmitted in mammals. Publication of both studies had been delayed many months due to fears that the research could be misused and become a bio-security threat.
Although these particular engineered forms of H5N1 have not been found in nature, the virus has potential to mutate enough such that it could become airborne.
H5N1 influenza can be deadly to people, but in its natural forms it does not easily transfer between people through respiratory droplets, as far as scientists know. The World Health Organization has recorded 355 humans deaths from it out of 602 cases, although some research has questioned this high mortality rate.
The journals Science and Nature had agreed to postpone the publication of the two studies related to the genetically altered virus.
In January, the National Science Advisory Board for Biosecurity recommended that this research be published without “methods or details” that terrorists might be able to use for biological weapons. The board also said the data could assist in preparing for a possible future outbreak, however.
Then in February, the World Health Organization convened a meeting, at which the recommendation was to publish the studies – just not yet. In April, the National Institutes of Health chimed in, also recommending publication.
The first study to be published on the topic was in the journal Nature, and was led by the University of Wisconsin-Madison researcher Yoshihiro Kawaoka. It was released in May.
The other research group, which authored the new study in Science, was led by Ron Fouchier at the Erasmus Medical Center in Rotterdam, Netherlands.
Both Kawaoka and Fouchier’s groups created a mutated version of H5N1 that made it easier to transmit from mammal to mammal. They used ferrets because these animals are a good approximation for how viruses behave in humans.
Fouchier’s study examines what mutations would be necessary to get the virus airborne. He and colleagues found five mutations consistent in a form of the H5N1 flu virus that could spread among ferrets through the air.
None of the ferrets died after developing the flu, the researchers said.
In a separate analysis, researchers looked at the likelihood that an airborne avian flu virus would evolve on its own from the H5N1 currently found in nature.
This study, also published in Science this week, looked at nearly 4,000 strains of influenza virus and frequently found two of the five mutations that appear to be involved in airborne transmission. These two mutations have been found in viruses from both birds and humans, although not in naturally-occurring H5N1 strains.
Derek Smith of the University of Cambridge, who co-authored that study, said at a press briefing that it’s possible that only three mutations are necessary for the virus to evolve.
Smith’s group also did mathematical modeling to look at whether the other mutations could evolve when the bird flu jumps to a human or other mammal.
“We find that it is possible for such a virus to evolve three mutations within a single host,” Smith said during the press call.
If it takes four for five mutations to become airborne, that would be more difficult – but it’s unclear just how likely it would be, Smith said.
While the Nature study looked at how a bird flu virus could become airborne through mutations and re-assortment with other viruses, the latest research in Science suggests mutations alone could do the trick.
Dr. Anthony Fauci, director of the National Institute of Allergy and Infectious Diseases, told reporters that the benefits from the Science study, in terms stimulating ideas and pursuing ways to understand the transmissibility, adaptability and pathogenesis of the virus, outweigh the risks that someone will use the data for nefarious purposes.
“Does that mean that there’s no risk? No, of course not. I can’t tell you at all
that there’s no risk. But the benefits in my mind outweigh the risks,” he said.
Making the research available generally will hopefully spark input on this topic from researchers in a wide variety of fields, he said.
It is technologically possible to create vaccine based on the genetic code of a flu virus strain including this one, researchers said. Several companies are already making H5N1 vaccines.
Research is ongoing to accelerate the amount of vaccine doses available by using adjuvants, which are agents that modify the effects of vaccines, Fauci said. There is also work ongoing into using computational sequencing to anticipate every possible influenza strain that could emerge, such that a databank could be established to prepare for the outbreak of any one of them, he said.
“Right now we’re in a much, much better position than we were when we had vaccine available after the peak of the 2009 H1N1 two years ago,” Fauci said.
How a Zombie Outbreak Could Happen in Real Life
Could zombies actually exist? What would it take for human corpses to rise up and hunt the living? We often think zombies are scientifically impossible — but actually, they’re just very implausible. Here’s one wayThe Walking Dead could happen in real life.
To start our zombie thought experiment, we need to make some basic assumptions. First, we’re ignoring all supernatural zombie origins. We’re also going to set aside space radiation, mysterious comets, or Russian satellites. Our focus will be narrowed to biological origins –- a zombie contagion. Of course, there are many different zombie scenarios in books and film, and no one theory is going to cover all of them perfectly.
The first aspect of human zombification we need to tackle is basic zombie physiology. In virtually every zombie scenario, zombies are able to function despite increasing levels of physical deterioration due to injury or decomposition. There has to be some mechanism for transmitting neural impulses from the brain to various body parts, and for providing energy to muscles so they can keep operating.
The most common science fictional explanation for zombie outbreaks is a virus — but viruses and bacterial infections are not known for building large new physical structures within the body. So let’s count viruses out. Instead, the need for a mechanism to activate deteriorating body parts actually provides the cornerstone of what is, in my opinion, the strongest theory: fungal infection.
We know that fungi can infect humans. We also know that fungal networks exist in most of the world’s forests. These mycorrhizal networks have a symbiotic relationship with trees and other plants in the forest, exchanging nutrients for mutual benefit. These networks can be quite large, and there are studies that demonstrate the potential for chemical signals to be transmitted from one plant to another via the mycorrhizal network. That, in turn, means that fungal filaments could perform both vascular and neural functions within a corpse.
This leads us to the following scenario: microscopic spores are inhaled, ingested, or transmitted via zombie bite. The spores are eventually dispersed throughout the body via the bloodstream. Then they lie dormant. When the host dies, chemical signals (or, more accurately, the absence of chemical signals) within the body that occur upon death trigger the spores to activate, and begin growing. The ensuing fungal network carries nutrients to muscles in the absence of respiration or normal metabolism.
Part of the fungal network grows within the brain, where it interfaces with the medulla and cerebellum, as well as parts of the brain involving vision, hearing and possibly scent. Chemicals released by the fungi activate basic responses within these brain areas. The fungi/brain interface is able to convert the electrochemical signals of neurons into chemical signals that can be transmitted along the fungal network that extends through much of the body. This signal method is slow and imperfect, which results in the uncoordinated movements of zombies. And this reliance on the host’s brain accounts for the “headshot” phenomenon, in which grievous wounds to the brain or spine seem to render zombies fully inert.
This leaves the problem of zombie metabolism. Where do the zombies get the nutrients needed to perform physical activity, plus the necessary nutrients to fuel the life-cycle of the fungi? This is most easily explained by the zombies’ constant, endless drive to devour meat. The fungal network would still need some way to metabolize meat, and zombies seem to be able to function even in the absence of a human digestive system.
It is possible that this particular fungi has evolved a means to extract energy and nutrients from meat in a similar manner to carnivorous plants. The ingestion of meat may actually be vestigial, an unintended result of the drive to bite. In this case, the fungi may draw energy from the decomposition of the host’s own organic material, which effectively puts a shelf-life on zombies (in addition to the deterioration of body structures beyond the point where the fungal network can compensate).
Accounts of dismembered parts moving purposefully may be apocryphal.
Now we have established a working theory for fungal zombies. How could such a disease arise? The goal of any biological organism is to live long enough to reproduce, but many pathogens are self-limited by their own lethality. The host dies before it has a chance to spread the pathogen inadvertently. This gives us two pathways for development of the zombie fungus. First, a fungal species existed that used the digestive tracts of mammals to travel. In other words, animals ingested the fungus, including spores. The spores were later defecated out in a new location. Some mutations occurred that caused the spores to gestate while still within the host. However, in most cases, the host’s immune system would destroy the fungus. Further mutations could lead to spores that only trigger once the host has died, avoiding this problem.
Another possibility is a fungal infection that was highly aggressive and caused rapid death within the host. That strain was not able to successfully reproduce as often as a mutated strain that delayed activation until post-mortem.
Of course, it’s one thing for a fungus to activate after the host dies, and quite another for the dead host to stand up and start attacking things. There are many evolutionary steps in between, which is why a zoonotic origin seems likely.
The precursor fungus could have been ingested by pigs, which are omnivorous. Captive pig populations, subject to overcrowding, would have been perfect places for the fungus to spread and mutate. In some poorly managed pig farms, dead pigs may have gone unnoticed, allowing post-mortem development of the fungus. Dead pigs were likely partially eaten by their living counterparts, allowing the fungal strains with post-mortem mutations to spread back into the population. The method of transfer from the pig population to the human population seems fairly obvious.
The evolution of fully mobile dead pigs probably started with a simple bite reflex that could transmit spores to nearby pigs. A bite combined with a muscular spasm, a sort of lunge, would work even better. After many generations, this developed into full post-mortem mobility. Thus, a dead host went from a drawback to an advantage, becoming a mobile platform for spore distribution. In fact, the zombie hunger drive may have originated as a spore distribution method –- only later was the ability to metabolize meat acquired. We can extrapolate this development to assume the further refinement of the fungal neural system, allowing for zombies which are far more coordinated and can run at nearly full speed.
While this type of behavior modification may seem unlikely, there is precedent for it within the animal world. Several species of parasitic wasps are able to reprogram the behavioral patterns of their hosts (bees, ants and even caterpillars), creating complex new behaviors beneficial to the wasp and detrimental to the host. While the hosts in these cases aren’t dead, this does demonstrate that complex chemical overrides can evolve in nature.
Hopefully scientists can develop an effective zombie fungicide in time.